Atypical hemolytic uremic syndrome (aHUS) is certainly associated with defective complement rules. by lengthy homologous repeats with lengthy interspersed nuclear components (retrotransposons) and we claim that non-allelic homologous recombination between these repeats leads to the increased loss of both genes. Impaired security of erythrocytes from enhance activation is seen in the serum of aHUS sufferers… Continue reading Atypical hemolytic uremic syndrome (aHUS) is certainly associated with defective complement